参考文献
[1]Arturson G.The pathophysiology of severe thermal injury.J Burn Care & Rehab,1985,6:129-146.
[2]Arturson G.Microvascular permeability of macromolecules in thermal injury.Acta Physiol Scand,1979,463(Suppl):111-122.
[3]Alexander F,et al.Arachidonlc acid metabolites mediated early burn edema.J Trauma,1984,24:709.
[4]Bruce M,Achauer(ed).Management of the Burned Patients.Appleton & Lange,Norwalk,Conne cticut,1987
[5]Bjork J,del Maestro RF,Arfor KE.Evidence for participation of hydroxyl radical in increased microvascular permeability.Agents Actions,1988,7(suppl):208.
[6]Braquet M,Carsin H,Guilbaud J,et al.Plasma atrial natriuretic peptide in severe tyermal injury.Lancet,1986,2:246.
[7]Bjerkes R,Vindenes H,Lacrum OD.Altered neutrophil functions in patients with large burns.Blood Cells,1990,16:127-141.
[8]Cochrance CG.The enhancement of inflamatory injury.Rev Respir Dis,1987,136:1-2.
[9]Dolecdk R,Moltni A,Brizio-Molteni L,et al(eds).Endoceinology of Burns.Philadelphia,Lea &Febiger,1990.
[10]Demling RH,Lalonde C.Early Post burn lipid peroxidation(effect ibuprofen and allopurinol).Surgery,1990,107:88-93.
[11]Davies JWL.Physiology Response to Burning Injury.Academic Press,London,1982.
[12]Falaky MH,Abdel Hafoz AH.Phygocytic activity of polymorphonuclear leucocytes in burns.Burns,1985,11:185-191.
[13]Friedl H,Till G,Trentz O,et al.Roles of histamine,complement and xanthine oxidne oxidase in thermal injury in skin.Am J Pathol,1989,135:203-210.
[14]Fang Zhi-yang,Sheng Zhi-youg,Li Ngao,et al(eds).Modern Treatment of Severe Burns.Springer-Verlag,Berlin,Heidelberg,Germany,1992.(https://www.daowen.com)
[15]Holady WG,John W.Cardiovascular effects of end ogenous opiate system.Annu Rev Pharmacol Toxicol,1983,23:541-594.
[16]Lefer AW.Interaction between myocardial depressant factor and vasoactive mediators with ischemia and shock.Amer J Physiol,1987,252:R193-R205.
[17]Muytinck H,Xavier J,Offerman W,et al.Whole body inflammation in trauma patients.Arch Surg,1998,123:1519-1523.
[18]Oldham K,Giuce K,Till G,et al.Actiuation of complemsnt by hydroxyl radical in thermal injury.Surgery,1985,104:272-279.
[19]Sasaki J,Cottam G,Baxter C.Lipid peroxidation following thermal injury.J Burn Care & Rehab,1987,4:251-254.
[20]Schiavon M,Landro DD,Baldo M,et al.A study of ranal damage in seriously burned patients.Burns,1988,14:107-114.
[21]Wilmore DN.Catecholamines,mediatos of hypermetabolic response to thermal injury.Ann Surg,1974,180:653-600.
[22]Wilmore DW.Hormonal responses and their effect on metabolism.Surg Clin North Am,1976,56:999.
[23]Yarbrough DⅢ.Pathophysiology of the Burn Wound.In Wagner M(ed):Care of the Burn-Injured Patient.Littleton,Mass.PSG Publishing,1981,21.
[24]Zawacki B.Reversal of capillary stasis and prevention of necrosis in burns.Ann Surg,1974,180:98.
[25]方之扬,吴中立,高学书,等.烧伤理论与实践.沈阳:辽宁科学技术出版社,1989.
[26]杨之骏,许伟石,史济湘.烧伤治疗.第2版.上海:上海科学技术出版社,1985.
[27]魏文汉.病理生理学.上海:上海科学技术出版社,1984.
[28]杨宗城.烧伤后血管内皮细胞损伤及其在早期脏器损害中的作用.中华烧伤杂志,2001,17(3):133-135.