参考文献

参考文献

[1]Jayaraman J, Jesudoss V A S, Menon V P, et al. Anti-inflammatory role of naringenin in rats with ethanol induced liver injury [J]. Toxicology Mechanisms and Methods, 2012, 22(7): 568-576.

[2]黄晓燕, 严杰, 李淑萍. 结核分枝杆菌感染细胞的信号传导通路研究进展[J]. 中国生物制品学杂志, 2006, 19(3): 323-325.

[3]KARACA N, ÖZTüRK G. Dermatolojide Toll-Benzeri Reseptörler[J]. Türkiye Klinikleri Dermatoloji Dergisi, 2012, 22(1): 8-18.

[4]Méndez-Samperio P. Role of interleukin-12 family cytokines in the cellular response to mycobacterial disease [J]. International Journal of Infectious Diseases, 2010, 14(5): e366-e371.

[5]Zhang Q, Lenardo M J, Baltimore D. 30 years of NF-κB: a blossoming of relevance to human pathobiology[J]. Cell, 2017, 168(1-2): 37-57.

[6]Albanese C, Wu K, D’Amico M, et al. IKKα regulates mitogenic signaling through transcriptional induction of cyclin D1 via Tcf[J]. Molecular biology of the cell, 2003, 14(2): 585-599.

[7]Potoyan D A, Zheng W, Komives E A, et al. Molecular stripping in the NFκB/IκB/DNA genetic regulatory network [J]. Proceedings of the National Academy of Sciences, 2016, 113(1): 110-115.

[8]Sasaki Y, Iwai K. Roles of the NF-κB pathway in B-lymphocyte biology[M]//B Cell Receptor Signaling. Springer, Cham, 2015: 177-209.

[9]Derudder E, Herzog S, Labi V, et al. Canonical NF-κB signaling is uniquely required for the long-term persistence of functional mature B cells[J].Proceedings of the National Academy of Sciences, 2016, 113(18): 5065-5070.

[10]Hayden M S, Ghosh S. NF-κB in immunobiology[J]. Cell research, 2011, 21(2): 223-244.

[11]Sun S C. The noncanonical NF-κB pathway[J]. Immunological reviews, 2012,246(1): 125-140.

[12]Xiao G, Fong A, Sun S C. Induction of p100 processing by NF-kappaBinducing kinase involves docking IKKalpha to p100 and IKKalpha-mediated phosphorylation[J].Journal of Biological Chemistry,2004.279(29):30099-105.

[13]Paul A, Edwards J, Pepper C, et al. Inhibitory-κB Kinase (IKK)α and nuclear factor-κB (NFκB)-inducing kinase (NIK)as anti-cancer drug targets[J]. Cells, 2018, 7(10): 176.

[14]Gray C M, Remouchamps C, McCorkell K A, et al. Noncanonical NF-κB signaling is limited by classical NF-κB activity[J]. Science signaling, 2014,7(311): ra13-ra13.

[15]D'Ignazio L, Bandarra D, Rocha S. NF-κB and HIF crosstalk in immune responses[J]. The FEBS journal, 2016, 283(3): 413-424.

[16]Paul M, Kemparaju K, Girish K S. Inhibition of constitutive NF-κB activity induces platelet apoptosis via ER stress [J]. Biochemical and biophysical research communications, 2017, 493(4): 1471-1477.

[17]Blanc L, Gilleron M, Prandi J, et al. Mycobacterium tuberculosis inhibits human innate immune responses via the production of TLR2 antagonist glycolipids[J]. Proceedings of the National Academy of Sciences, 2017, 114(42): 11205-11210.

[18]周敬禹, 杨桂连, 王春凤. TLR 信号途径对于NF-κB 活化的调控及作用机制[J]. 畜牧与兽医, 2015(1): 117-120.

[19]Fallahi-Sichani M, Kirschner D E, Linderman J J. NF-κB signaling dynamics play a key role in infection control in tuberculosis[J]. Frontiers in physiology,2012, 3: 170.

[20]Juffermans N P, Verbon A, van Deventer S J H, et al. Elevated chemokine concentrations in sera of human immunodeficiency virus (HIV)-seropositive and HIV -seronegative patients with tuberculosis: a possible role for mycobacterial lipoarabinomannan[J]. Infection and immunity, 1999, 67(8):4295-4297.

[21]Tang N L S, Fan H P Y, Chang K C, et al. Genetic association between a chemokine gene CXCL-10 (IP-10, interferon gamma inducible protein 10)and susceptibility to tuberculosis[J]. Clinica chimica acta, 2009, 406(1-2):98-102.

[22]Wang J, Li B X, Ge P P, et al. Mycobacterium tuberculosis suppresses innate immunity by coopting the host ubiquitin system[J]. Nature immunology, 2015,16(3): 237-245.

[23]Zhang H, Ouyang H, Wang D, et al. Mycobacterium tuberculosis Rv2185c contributes to nuclear factor-κB activation[J]. Molecular immunology, 2015,66(2): 147-153.

[24]Kumar M, Sahu S K, Kumar R, et al. MicroRNA let-7 modulates the immune response to Mycobacterium tuberculosis infection via control of A20, an inhibitor of the NF-κB pathway[J].Cell host&microbe,2015,17(3):345-356.

[25]Clevers H, Nusse R. Wnt/β-catenin signaling and disease[J]. Cell, 2012, 149(6): 1192-1205.

[26]Jang J, Jung Y, Chae S, et al. Wnt/β-catenin pathway modulates the tnf-αinduced inflammatory response in bronchial epithelial cells[J]. Biochemical and biophysical research communications, 2017, 484(2): 442-449.

[27]Schön S, Flierman I, Ofner A, et al. β‐catenin regulates NF‐κB activity via TNFRSF19 in colorectal cancer cells [J]. International journal of cancer,2014, 135(8): 1800-1811.

[28]Deng J, Miller S A, Wang H Y, et al. β-catenin interacts with and inhibits NF-κB in human colon and breast cancer[J].Cancer cell,2002,2(4):323-334.

[29]Nejak-Bowen K, Kikuchi A, Monga S P S. Beta-catenin-NF-κB interactions in murine hepatocytes: A complex to die for[J]. Hepatology, 2013, 57(2):763-774.

[30]Pei J, Fan L, Nan K, et al. Excessive activation of TLR4/NF-κB interactively suppresses the canonical Wnt/β-catenin pathway and induces SANFH in SD rats[J]. Scientific reports, 2017, 7(1): 1-13.

[31]Tian, B., et al., The NFkappaB subunit RELA is a master transcriptional regulator of the committed epithelial -mesenchymal transition in airway epithelial cells. J Biol Chem, 2018. 293(42): 16528-16545.

[32]Du X, Poltorak A, Wei Y, et al. Three novel mammalian toll-like receptors:gene structure, expression, and evolution [J]. European cytokine network,2000, 11(3): 362-71.

[33]Janssens S, Beyaert R. Role of Toll-like receptors in pathogen recognition[J].Clinical microbiology reviews, 2003, 16(4): 637-646.

[34]Kawai T, Akira S. Pathogen recognition with Toll-like receptors[J]. Current opinion in immunology, 2005, 17(4): 338-344.

[35]Hartman Z C, Kiang A, Everett R S, et al. Adenovirus infection triggers a rapid, MyD88-regulated transcriptome response critical to acute-phase and adaptive immune responses in vivo[J]. Journal of virology, 2007, 81(4):1796-1812.

[36]Li Y, Shi J, Yang J, et al. A Wnt/β-catenin negative feedback loop represses TLR -triggered inflammatory responses in alveolar epithelial cells[J].Molecular immunology, 2014, 59(2): 128-135.

[37]Silva-García O, Valdez-Alarcón J J, Baizabal-Aguirre V M. Wnt/β-catenin signaling as a molecular target by pathogenic bacteria [J]. Frontiers in immunology, 2019, 10: 2135.

[38]Villaseñor T, Madrid-Paulino E, Maldonado-Bravo R, et al. Activation of the Wnt pathway by Mycobacterium tuberculosis: a Wnt–Wnt situation[J].Frontiers in immunology, 2017, 8: 50.

[39]Wu X, Deng G, Li M, et al. Wnt/β-Catenin signaling reduces Bacillus Calmette-Guerin-induced macrophage necrosis through a ROS-mediated PARP/AIF-dependent pathway[J]. BMC immunology, 2015, 16(1): 16.

[40]Wu X, Deng G, Hao X, et al. A caspase-dependent pathway is involved in Wnt/β-catenin signaling promoted apoptosis in Bacillus Calmette-Guerin infected RAW264. 7 macrophages [J]. International journal of molecular sciences, 2014, 15(3): 5045-5062.

[41]Wu X, Zhang J, Ma C, et al. A role for Wnt/β-catenin signalling in suppressing Bacillus Calmette-Guerin-induced macrophage autophagy [J]. Microbial pathogenesis, 2019, 127: 277-287.

[42]Schaale K,Brandenburg J,Kispert A,et al.Wnt6 is expressed in granulomatous lesions of Mycobacterium tuberculosis -infected mice and is involved in macrophage differentiation and proliferation[J]. The Journal of Immunology,2013, 191(10): 5182-5195.

[43]Schaale K, Neumann J, Schneider D, et al. Wnt signaling in macrophages:augmenting and inhibiting mycobacteria-induced inflammatory responses[J].European journal of cell biology, 2011, 90(6-7): 553-559.?