参考文献

参考文献

[1]DJEBALI S, et al. Landscape of transcription in human cells[J]. Nature, 2012.

[2]SALTA E, et al. RNAs with essential roles in neurodegenerative disorders[J]. The Lancet Neurology, 2012.

[3]FATICA A, et al. Long non-coding RNAs: new players in cell differentiation and development[J].Nature Reviews Genetics, 2013.

[4]DERRIEN T, et al. The GENCODE v7 catalog of human long noncoding RNAs: analysis of their gene structure, evolution, and expression[J]. Genome Res, 2012.

[5]CLARK M B, et al. Genome-wide analysisof long noncoding RNA stability[J]. Genome Res, 2012.

[6]BENTWICH I, et al. Identification of hundreds of conserved and nonconserved human microRNAs[J]. Nat Genet, 2005.

[7]BIRNEY E, et al. Identification and analysis of functional elements in 1% of the human genome by the ENCODE pilot project[J]. Nature, 2007.

[8]LIU B, et al. A Cytoplasmic NF- x Binteracting Long Noncoding RNA Blocksix B Phosphorylation and Suppresses Breast Cancer Metastasis[J]. Cancer Cell, 2015.

[9]GUPTA R A, et al. Long non-coding RNA HOTAiR reprograms chromatin state to promote cancer metastasis[J]. Nature, 2010.

[10]SORENSEN K P, et al. Long non-coding RNA HOTAIRs is an independent prognostic marker of metastasis in estrogen receptor-positive primary breast cancer[J]. Breast Cancer Research and Treatment, 2013.

[11]BHAN A, et al. Antisense Transcript Long Noncoding RNA (lncRNA) HOTAIR is Transcriptionally Induced by Estradiol[J]. Journal of Molecular Biology, 2013.

[12]CHIYOMARU T, et al. Long non-coding RNA HOTAIR is targeted and regulated by miR-141 in human cancer cells[J]. Journal of Biological Chemistry, 2014.

[13]BERTEAUX N, et al. H19 mRNA-like noncoding RNA promotes breast cancer cell proliferation through positive control by E2F1[J]. J Biol Chem, 2005.

[14]LOTTIN S, et al. Overexpression of an ectopic H19 gene enhances the tumorigenic properties of breast cancer cells[J]. Carcinogenesis, 2002.

[15]SUN H, et al. H19 lncRNA mediates 17 beta-estradiol-induced cell proliferation in MCF-7 breast cancer cells[J]. Oncol Rep, 2015.

[16]HAO Y, et al. Tumour-suppressor activity of H19 RNA[J]. Nature, 1993.

[17]YOSHIMIZU T, et al. The H19 locus acts in vivo as a tumor suppressor[J]. Proc Natl Acad Sci U S A, 2008.

[18]KAWAKAMI T, et al. Characterization of loss-of-inactive X in Klinefelter syndrome and female-derived cancer cells[J]. Oncogene, 2004.

[19]BENOIT M H, et al. Global analysis of chromosome X gene expression in primary cultures of normal ovarian surface epithelial[J]. Int.JOncol, 2007.

[20]GANESAN S, et al. BRCA1 supports XIST RNA concentration on the inactive X chromosome[J]. Cell, 2002.

[21]ZHANG Z, et al. Negative regulation of lncRNA GAS5 by miR-21[J]. Cell Death Differ, 2013.

[22]PICKARD M R, et al. Regulation of apoptosis by long non-coding CTASS in breast cancer cells: implications for chemotherapy[J]. Breast Cancer Research and Treatment, 2014.

[23]SILVA J M, et al. LSINCT5 is over expressed in breast and ovarian cancer and affects cellular proliferation[J]. RNA Biology, 2014.

[24]CHEN W, et al. Expression of neural BC200 RNA in human tumours[J]. J Pathol, 1997.

[25]Iacoangeli A, et al. BC200 RNA in invasive and preinvasive breast cancer[J]. Carcinogenesis, 2004.

[26]GODINHO M, et al. Characterization of BCAR4, a novel oncogene causing endocrine resistance in human breast cancer cells[J]. Journal of Cellular Physiology, 2011.

[27]GODINHO M F, et al. Relevance of BCAR4 in tamoxifen resistance and tumour aggressiveness of human breast cancer[J]. Br. J Cancer, 2010.

[28]JIANG M, et al. A novel long non-coding RNA-ARA: Adriamycin Resistance Associated [J]. Biochemical Pharmacology, 2014,